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First, inflammation suppresses the hypothalamus-pituitary-thyroid (HPT) axis.

One study showed a single injection of the inflammatory cytokine TNF-alpha reduced blood levels of TSH, T3, free T4, free T3 and TRH for 5 days.

In the last post I explained that, for the vast majority of patients, hypothyroidism is an autoimmune disease. It’s the reason both conventional and alternative treatments are so often ineffective.In this post I’m going to show you why taking replacement thyroid hormones without addressing the underlying immune imbalance is doomed to fail. Once again the conventional approach falls short because it ignores the underlying cause of the problem.Before I sign off, I just want to make one thing clear.I’m not saying thyroid medication isn’t necessary or useful.In fact, I think it’s an important part of treating Hashimoto’s – especially when TSH is consistently elevated and T4 and T3 are consistently low.

My point is thyroid medication is only one piece of the puzzle, and it won’t be effective on its own unless the autoimmunity and inflammation are addressed.

Patients who don’t convert T4 to T3 well do better on bio-identical hormones like Armour, because it contains both T4 and T3 (in a 4.22:1 ratio).

Inflammation disrupts thyroid metabolism in several other ways, but I think these three examples make the point. Inflammation causes HPT axis disruption, decreased receptor function, and decreased conversion of T4 to T3.

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